10 g Speed Sulfate 90%

Buy Speed Sulfate Online

Buy Speed Sulfate Online This is listing is FE. That means I receive my money upfront.

Sent in a flat hardbox envelope without tracking.
We ship multiple times a week for fast processing.
You may contact me for a custom listing.

Negative reviews receive no support. Communicate your needs. No need for negative energy.

Speed Sulfate is a lot more potent than regular speed paste. Consume with caution. Start with 10mg and go from there.

Amphetamine is a stimulant substance of the phenethylamine class. Chemically, it is the parent compound of the substituted amphetamines, a group which includes a diverse range of substances. Amphetamine acts by promoting the release of the neurotransmitters dopamine and norepinephrine in the brain.

Amphetamine was first synthesized in 1887, but its psychostimulant effects were not discovered until 1929. In the 1930s, it began to be marketed and sold over-the-counter under the name Benzedrine. It was widely used to treat a wide range of ailments such as alcohol hangover, narcolepsy, depression, and weight reduction. Today, amphetamine is used clinically for the treatment of attention deficit hyperactivity disorder , narcolepsy, and obesity.
Additionally, it sees widespread illicit use as a performance enhancing agent and recreational substance.

Amphetamine is considered to be the quintessential member of the stimulant class due to its extensively studied effects and long history of clinical use. Subjective effects include stimulation, focus enhancement, motivation enhancement, increased libido, appetite suppression, and euphoria. It is usually taken orally, but can also be insufflated, injected, or administered rectally. Lower doses tend to increase focus and productivity while higher doses tend to increase sociability, sexual desire, and euphoria.

Recreational use of amphetamine is associated with abuse and dependence. It is highly advised to use harm reduction practices if using this substance.

EFFECTS

Amphetamine is reported to be very energetic and stimulating. It can encourage physical activities such as dancing, socializing, running, or cleaning. The particular style of stimulation which amphetamine produces can be described as forced. This means that at higher dosages, it becomes difficult or impossible to keep still. Jaw clenching, involuntary bodily shakes, and vibrations become present, resulting in extreme shaking of the entire body, unsteadiness of the hands, and a general loss of fine motor control. This is replaced with mild fatigue and general exhaustion during the offset of the experience.

The body high of amphetamine can be described as a moderate euphoric tingling sensation that encompasses the entire body. This sensation maintains a consistent presence that steadily rises with the onset and hits its limit once the peak has been reached.

The visual effects of amphetamine are inconsistent and occur only mildly noticeable at higher doses. They are somewhat comparable to deliriant visuals and occur more readily in darker areas.

Hallucinations occur very rarely, and typically only when the user has taken high doses, is coming down, or has been awake for unusually long periods. They are usually very mild when they do occur.

AFTER EFFECTS

The effects which occur during the offset of a stimulant experience generally feel negative and uncomfortable in comparison to the effects which occurred during its peak. This is often referred to as a comedown and occurs because of neurotransmitter depletion. Its effects commonly include anxiety, increased heart rate, wakefulness, lack of motivation

RISKS

As of March 2014, there is no evidence that amphetamine is directly neurotoxic in humans. However, high-dose amphetamine can cause indirect neurotoxicity as a result of increased oxidative stress from reactive oxygen species and autoxidation of dopamine.

In rodents and primates, sufficiently high doses of amphetamine causes damage to dopamine neurons, characterized as reduced transporter and receptor function. Animal models of neurotoxicity from high-dose amphetamine exposure indicate that the occurrence of very high body temperature is necessary for the development of amphetamine-induced neurotoxicity.

Melatonin has been shown to prevent and reverse amphetamine induced neurotoxicity of TH-pSer40 and calpastatin levels in the Substantia Nigra of rats if used 30min+ before dosing.

It is strongly recommended that one use harm reduction practices when using this substance.

The LD50 (the dosage required to kill 50% of the test subjects) of amphetamine in rats has been found to be between roughly 15 mg and 180 mg per kilogram, depending on the study.

Amphetamine has a high abuse potential and can cause dependence with chronic use. When dependence has developed, cravings and withdrawal effects may occur if use is suddenly discontinued. Withdrawal symptoms include paranoia, depression, dream potentiation, anxiety, itching, mood swings, irritability, fatigue, insomnia, an intense craving for more amphetamine or other stimulants. Addiction is a serious risk with heavy recreational amphetamine use, but is unlikely to arise from typical medical use.

Tolerance to many of the effects of amphetamine develops with prolonged and repeated use. This results in users having to administer increasingly large doses to achieve the same effects. Upon single administration, it takes about 3 – 7 days for the tolerance to be reduced to half and 1 – 2 weeks to be back at baseline (in the absence of further consumption). Amphetamine presents cross-tolerance with all dopaminergic stimulants, meaning that after the consumption of amphetamine most stimulants will have a reduced effect.

Severe amphetamine overdose can result in a stimulant psychosis that may present with a variety of symptoms such as delusions, paranoia, hallucinations. A review on treatment for amphetamine abuse-induced psychosis states that about 5–15% of users fail to recover completely. The same review asserts that antipsychotic medications effectively resolve the symptoms of acute amphetamine psychosis. Psychosis very rarely arises from therapeutic use.

DANGEROUS INTERACTIONS

Alcohol – Drinking alcohol on stimulants is considered risky because it reduces the sedative effects of the alcohol that the body uses to gauge drunkenness. This often leads to excessive drinking with greatly reduced inhibitions, increasing the risk of liver damage and increased dehydration. The effects of stimulants will also allow one to drink past a point where they might normally pass out, increasing the risk. If you do decide to do this then you should set a limit of how much you will drink each hour and stick to it, bearing in mind that you will feel the alcohol and the stimulant less. Buy Speed Sulfate Online
GHB/GBL – Stimulants increase respiration rate allowing a higher dose of sedatives. If the stimulant wears off first then the depressant effects of the GHB/GBL may overcome the user and cause respiratory arrest.
Opioids – Stimulants increase respiration rate allowing a higher dose of opiates. If the stimulant wears off first then the opiate may overcome the patient and cause respiratory arrest.
Cocaine – This combination of stimulants will increase strain on the heart. It is not favored as cocaine has a mild blocking effect on dopamine releasers like amphetamine. To Purchase Speed Sulfate Online contact us